In distinction to neurodegerative diseases and cancer, the contribution of TG2 activity to celiac sprue pathogenesis has long been rather properly founded. The deamidation of precise glutamine residues specific by TG2 in gluten peptides is revealed to boost peptide binding to the illness-involved proteins HLA-DQ2/8, that are the only real https://luism766xis8.bcbloggers.com/profile